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Phospholipid hydroperoxides (PLOOHs) execute a critical role in ferroptosis under peroxidizing conditions, catalyzing and generating complex products including oxygen radicals, while ROS trigger cellular damage by promoting membrane PUFA peroxidation (41)

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To date, a variety of mitochondria-resident proteins, such as CDGSH iron sulfur domain 1 (CISD1), FUN14 domain containing 1 (FUNDC1) and acyl-CoA synthetase family member 2 (ACSF2), have been confirmed to participate in regulating ferroptosis and kidney diseases[48, 49]

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